Preparation of a 6FDA-DAM/ODA Mixed Matrix Membrane Doped with MOFs and Its Application in Gas Separation.

Mixed matrix membranes (MMMs) can significantly improve gas separation performance, but the type and state of the filler in the membrane matrix are key indicators for the development of MMMs. Therefore, in this work, 6FDA-DAM/ODA (1:1), metal-organic frameworks (MOFs) with different particle sizes (UiO-66 and UiO-66-NH) were synthesized, and then MOFs were doped into 6FDA-DAM/ODA to prepare MMMs. The effects of the dopant materials and their particle sizes on the gas separation performance of the membranes were investigated by testing the permeability of the MMMs to H, CO, CH, and N.

Powdery Mildew of Caused by in China.

Echinacea purpurea (Eastern Purple Coneflower) is a perennial herbaceous plant belonging to the Asteraceae. It originated from North America and is cultivated all over the world. Extracts of E.

Engineering Oxygen Intermediates Adsorption on Amorphous NiFe Alloys for Highly Active and Selective Electrochemical Biomass Conversion.

Electrochemical 5-hydroxymethylfurfural (HMF) oxidation reaction (HMFOR) offers a promising route to transform biomass into value-added chemicals. However, the competing oxygen evolution reaction (OER) greatly limits the HMFOR selectivity. Herein, we report a facile doping strategy to engineer oxygen intermediates adsorption on amorphous NiFe alloys to boost highly selective electrochemical HMF oxidation to produce 2,5-furandicarboxylic acid (FDCA), among which, amorphous Mn-doped NiFeB alloy displays a low HMFOR onset potential of 1.

Mitochondrial transplantation for the treatment of cardiac and noncardiac diseases: mechanisms, prospective, and challenges.

Mitochondrial transplantation (MT) is a promising therapeutic strategy that involves introducing healthy mitochondria into damaged tissues to restore cellular function. This approach has shown promise in treating cardiac diseases, such as ischemia-reperfusion injury, myocardial infarction, and heart failure, where mitochondrial dysfunction plays a crucial role. Transplanting healthy mitochondria into affected cardiac tissue has resulted in improved cardiac function, reduced infract size, and enhanced cell survival in preclinical studies.