Publications by authors named "F PAULINO"

Article Synopsis
  • The study aims to assess how COPD (Chronic Obstructive Pulmonary Disease) affects the efficiency of respiratory and peripheral muscles during exercise.
  • Healthy subjects showed significantly better respiratory and peripheral neuromuscular efficiency than COPD patients across various exercise intensities.
  • The findings indicate that dynamic hyperinflation negatively impacts both respiratory and peripheral muscle efficiency in COPD patients.
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Background: Youth sports specialization has become more prevalent despite consequences such as increased injury rates and burnout. Young athletes, coaches, and parents continue to have misconceptions about the necessity of sports specialization, giving athletes the encouragement to focus on a single sport at a younger age.

Purpose: To characterize the motivations for specialization and determine when elite athletes in various individual and team sports made the decision to specialize.

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Background: This study examined the immediate outcomes during the perioperative period associated with drains in the setting of total shoulder arthroplasty or reverse shoulder arthroplasty. We hypothesized that drain use would result in lower postoperative hemoglobin and hematocrit levels that would increase transfusion rates and longer hospital stays that would increase hospital costs.

Methods: The study prospectively randomized 100 patients (55% women; average age, 69.

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Cellulose nanowhiskers (CNWs, 90% crystalline) were used to enhance the adsorption capacity of chitosan-g-poly(acrylic acid) hydrogel. The composites up to 20w/w-% CNWs showed improved adsorption capacity towards methylene blue (MB) as compared to the pristine hydrogel. At 5w/w-% CNWs the composite presented the highest adsorption capacity (1968mg/g).

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Staphylococcus aureus triggers inflammation through inflammasome activation and recruitment of neutrophils, responses that are critical for pathogen clearance but are associated with substantial tissue damage. We postulated that necroptosis, cell death mediated by the RIPK1/RIPK3/MLKL pathway, would function to limit pathological inflammation. In models of skin infection or sepsis, Mlkl-/- mice had high bacterial loads, an inability to limit interleukin-1b (IL-1b) production, and excessive inflammation.

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