Publications by authors named "F Daussin"

This study aimed to assess the physiological responses to repeated running exercise performed at supramaximal intensity and with end-expiratory breath holding (EEBH) up to the breaking point. Eight male runners participated in two running testing sessions on a motorized treadmill. In the first session, participants performed two sets of 8 repetitions at 125% of maximal aerobic velocity and with maximum EEBH.

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Hingrand, C, Olivier, N, Combes, A, Bensaid, S, and Daussin, FN. Power is more relevant than ascensional speed to determine metabolic demand at different gradient slopes during running. J Strength Cond Res 37(11): 2298-2301, 2023-Trail running is characterized by successive uphill and downhill running sessions.

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Article Synopsis
  • - Sepsis-induced myopathy leads to muscle fiber atrophy and mitochondrial issues, impacting recovery outcomes, but the role of overall energy deficit in this process was not previously studied.
  • - In the experiment, three mouse groups were analyzed: sepsis mice with reduced caloric intake, sham-fed mice, and sham mice fed in line with the sepsis group's intake, to determine energy balance and muscle characteristics.
  • - Results indicated that while sepsis caused a 17% reduction in muscle size and decreased mitochondrial function, the energy deficit alone did not fully explain the observed muscle issues, suggesting different metabolic adaptations in the sham pair-fed mice compared to the sepsis group.
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Introduction: The study aimed to interpret the evolution of the physical performance of rugby sevens and rugby union French international players from 2009 to 2020.

Methods: 631 players from the French national teams were divided into three groups: forwards, backs and sevens. The performances evaluated were anthropometric characteristics, strength tests (1 RM bench press and 1 RM pull-up), aerobic capacity (YoYo IR1 test) and speed tests (10 m, 20 m and 50 m).

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Protein O-GlcNAcylation is increasingly recognized as an important cellular regulatory mechanism, in multiple organs including the heart. However, the mechanisms leading to O-GlcNAcylation in mitochondria and the consequences on their function remain poorly understood. In this study, we use an in vitro reconstitution assay to characterize the intra-mitochondrial O-GlcNAc system without potential cytoplasmic confounding effects.

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