Brain Acetylcholinesterase and Blood Butyrylcholinesterase Levels in Carnaby's Cockatoos (Zanda latirostris) with Carnaby's Hindlimb Paralysis Syndrome (CHiPS).

Through evaluation of serum and plasma buterylcholinesterase (BChE) and brain acetylcholinesterase (AChE) activity, we investigated the possibility of the involvement of an acute organophosphate toxicosis in the pathogenesis of ongoing annual outbreaks of paresis and paralysis that in some cases progress to death, in endangered Western Australian Carnaby's cockatoos (Zanda latirostris). The condition, named Carnaby's hindlimb paralysis syndrome (CHiPS), was first described in 2012. Following initial investigations involving clinical, epidemiologic, toxicologic, gross necropsy, and histologic evaluation, a toxic etiology, specifically an organophosphate toxicosis, was considered most likely.

Distal radial osteochondroma causing expansile lysis and ulna fracture in a dog.

Objective: To describe the diagnosis, management, and outcome of a dog with a right distal radial osteochondroma that penetrated the ulna, causing expansile lysis and fracture.

Animal: A 9-month-old entire female German Shorthaired Pointer.

Clinical Presentation, Progression, And Procedures: The dog had a 2-month history of weight-bearing lameness of the right forelimb and a 2-week history of a progressively enlarging, firm swelling on the distolateral antebrachium.

Gross and histopathological findings in hindlimb paralysis syndrome in wild Carnaby's black cockatoos (Zanda laitirostris).

The aim of this study was to describe the gross and histopathological features of a neurological syndrome in endangered Western Australian Carnaby's black cockatoos (Zanda laitirostris) that was first observed in 2012. The syndrome, named hindlimb paralysis syndrome in Carnaby's cockatoos (CHiPS), is characterized by annual outbreaks of hindlimb paralysis with occasional loss of deep pain and cloacal tone, typically occurring between January and March. Previous limited investigations suggested a possible toxic aetiology.

Quantification of gastroesophageal regurgitation in brachycephalic dogs.

Background: Gastroesophageal reflux and regurgitation occurs in brachycephalic dogs, but objective assessment is lacking.

Objectives: Quantify reflux in brachycephalic dogs using an esophageal pH probe and determine the association with scored clinical observations.

Animals: Fifty-one brachycephalic dogs.