Analog reservoir computing via ferroelectric mixed phase boundary transistors.

Analog reservoir computing (ARC) systems have attracted attention owing to their efficiency in processing temporal information. However, the distinct functionalities of the system components pose challenges for hardware implementation. Herein, we report a fully integrated ARC system that leverages material versatility of the ferroelectric-to-mixed phase boundary (MPB) hafnium zirconium oxides integrated onto indium-gallium-zinc oxide thin-film transistors (TFTs).

Contention-Less Multi-Link Synchronous Transmission for Throughput Enhancement and Heterogeneous Fairness in Wi-Fi 7.

Multi-link operation (MLO) is a new and essential mechanism of IEEE 802.11be Extremely High Throughput (Wi-Fi 7) that can increase throughput and decrease latency in Wireless Local Area Networks (WLANs). The MLO enables a Multi-Link Device (MLD) to perform Simultaneous Transmission and Reception (STR) in different frequency bands.

Interference-Aware Two-Level Differentiated Transmission for Improving Downlink Spatial Reuse in Dense WLANs.

In this study, we address the problem of downlink throughput degradation in dense wireless local area networks (WLANs) based on the IEEE 802.11ax standard. We demonstrate that this problem essentially results from the asymmetric characteristic of carrier sense multiple access between downlink and uplink transmissions in infrastructure WLANs, and it is exacerbated by a dynamic sensitivity control algorithm that aims to improve spatial reuse (SR) in IEEE 802.

OFDMA Backoff Control Scheme for Improving Channel Efficiency in the Dynamic Network Environment of IEEE 802.11ax WLANs.

IEEE 802.11ax uplink orthogonal frequency division multiple access (OFDMA)-based random access (UORA) is a new feature for random channel access in wireless local area networks (WLANs). Similar to the legacy random access scheme in WLANs, UORA performs the OFDMA backoff (OBO) procedure to access the channel and decides on a random OBO counter within the OFDMA contention window (OCW) value.

FNDC-1-mediated mitophagy and ATFS-1 coordinate to protect against hypoxia-reoxygenation.

Mitochondrial quality control (MQC) balances organelle adaptation and elimination, and mechanistic crosstalk between the underlying molecular processes affects subsequent stress outcomes. FUNDC1 (FUN14 domain containing 1) is a mammalian mitophagy receptor that responds to hypoxia-reoxygenation (HR) stress. Here, we provide evidence that FNDC-1 is the ortholog of FUNDC1, and that its loss protects against injury in a worm model of HR.

An Efficient Relayed Broadcasting based on the Duplication Estimation Model for IoT Applications.

In this paper, we consider relay-based broadcasting in wireless ad hoc networks, which can enable various emerging services in the Internet of Things (IoT). In this kind of traffic dissemination scheme, also known as flooding, all the nodes not only receive frames but also rebroadcast them. However, without an appropriate relay suppression, a broadcast storm problem arises, i.

RPM-1 and DLK-1 regulate pioneer axon outgrowth by controlling Wnt signaling.

Axons must correctly reach their targets for proper nervous system function, although we do not fully understand the underlying mechanism, particularly for the first 'pioneer' axons. In , AVG is the first neuron to extend an axon along the ventral midline, and this pioneer axon facilitates the proper extension and guidance of follower axons that comprise the ventral nerve cord. Here, we show that the ubiquitin ligase RPM-1 prevents the overgrowth of the AVG axon by repressing the activity of the DLK-1/p38 MAPK pathway.

Medical-Grade Channel Access and Admission Control in 802.11e EDCA for Healthcare Applications.

In this paper, we deal with the problem of assuring medical-grade quality of service (QoS) for real-time medical applications in wireless healthcare systems based on IEEE 802.11e. Firstly, we show that the differentiated channel access of IEEE 802.

Anoxia-reoxygenation regulates mitochondrial dynamics through the hypoxia response pathway, SKN-1/Nrf, and stomatin-like protein STL-1/SLP-2.

Many aerobic organisms encounter oxygen-deprived environments and thus must have adaptive mechanisms to survive such stress. It is important to understand how mitochondria respond to oxygen deprivation given the critical role they play in using oxygen to generate cellular energy. Here we examine mitochondrial stress response in C.

Hypoxia regulates glutamate receptor trafficking through an HIF-independent mechanism.

Oxygen influences behaviour in many organisms, with low levels (hypoxia) having devastating consequences for neuron survival. How neurons respond physiologically to counter the effects of hypoxia is not fully understood. Here, we show that hypoxia regulates the trafficking of the glutamate receptor GLR-1 in C.

Structures of PHR domains from Mus musculus Phr1 (Mycbp2) explain the loss-of-function mutation (Gly1092-->Glu) of the C. elegans ortholog RPM-1.

PHR [PAM (protein associated with Myc)-HIW (Highwire)-RPM-1 (regulator of presynaptic morphology 1)] proteins are conserved, large multi-domain E3 ubiquitin ligases with modular architecture. PHR proteins presynaptically control synaptic growth and axon guidance and postsynaptically regulate endocytosis of glutamate receptors. Dysfunction of neuronal ubiquitin-mediated proteasomal degradation is implicated in various neurodegenerative diseases.

The ubiquitin ligase RPM-1 and the p38 MAPK PMK-3 regulate AMPA receptor trafficking.

Ubiquitination occurs at synapses, yet its role remains unclear. Previous studies demonstrated that the RPM-1 ubiquitin ligase organizes presynaptic boutons at neuromuscular junctions in C. elegans motorneurons.