Publications by authors named "Erika Foster"

Probation is the most common disposition for adjudicated youth, but little is known about which specific requirements are commonly imposed on juveniles, the requirements with which juveniles most often fail to comply, and how certain youth characteristics and/or imposed requirements might relate to probation noncompliance. An investigation of 120 archived files of youth represented by an urban public defender's office identified 29 probation requirements imposed on youth and 18 requirements with which youth commonly failed to comply. Results revealed that 52% of youth failed to comply with at least one probation requirement; prior probation noncompliance and race were both significantly associated with noncompliance in the examined probation disposition.

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  • Researchers have identified a mutation in the CACNA1C gene linked to Timothy Syndrome, a rare condition causing heart issues and physical abnormalities.
  • The study reveals that the calcium channel CaV1.2 is crucial for jaw development, as it is expressed in early structures that form the jaw.
  • Findings indicate that alterations in calcium signals through CaV1.2 lead to changes in jaw growth, providing new insights into the role of calcium channels in the development of nonexcitable cells.
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Potassium channel interacting proteins (KChIP) are Ca(2+)-binding proteins that originally were identified as auxiliary subunits for K(V)4 channels. K(V)4 channels encode the voltage gated A-current (I(A)) in neuronal tissue and the fast, transient outward current (I(to,f)) in cardiac tissue. Recently, we have reported that KChIP2 functionally modulates the cardiac Ca(V)1.

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  • NDRG4 is a lesser-known member of the tumor suppressive NDRG family, with expression primarily in the heart and brain unlike its relatives, NDRG1-3.
  • Research shows NDRG4 is highly expressed in glioblastoma multiforme (GBM) cells and is vital for the survival of both primary astrocytes and various GBM cell lines.
  • Knocking down NDRG4 leads to G(1) cell cycle arrest and apoptosis, reducing tumorigenic potential in GBM cells, marking its function as distinct from other NDRG family members.
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