Method for redistributing ill-defined causes of death.

Analysis of causes of death is crucial for monitoring an epidemiological situation and for developing adequate policy responses. However, the comparability of cause-specific mortality data depends on the proportion of ill-defined deaths. To eliminate the bias resulting from the varying proportions of such causes over time and between populations, deaths from ill-defined causes need to be reassigned to other categories.

Experimental factors influencing the bioaccessibility and the oxidative potential of transition metals from welding fumes.

Inhalation of welding fumes (WFs) containing high levels of transition metals (Cr, Cu, Fe, Mn, Ni…) is associated with numerous health effects including oxidative stress. However, the measurements of the oxidative potential (OP) and bioaccessibility of WF transition metals depend on several physicochemical parameters and may be subject to several experimental artifacts. In this work, we investigated the influence of the experimental conditions that may affect the bioaccessibility of transition metals and their OP on stainless-steel WF extracts.

Oxidative stress and inflammation induced by air pollution-derived PM persist in the lungs of mice after cessation of their sub-chronic exposure.

More than 7 million early deaths/year are attributable to air pollution. Current health concerns are especially focused on air pollution-derived particulate matter (PM). Although oxidative stress-induced airway inflammation is one of the main adverse outcome pathways triggered by air pollution-derived PM, the persistence of both these underlying mechanisms, even after exposure cessation, remained poorly studied.

Metal enriched quasi-ultrafine particles from stainless steel gas metal arc welding induced genetic and epigenetic alterations in BEAS-2B cells.

Recent evidence has supported welding fume (WF)-derived ultrafine particles (UFP) could be the driving force of their adverse health effects. However, UFP have not yet been extensively studied and are currently not included in present air quality standards/guidelines. Here, attention was focused on the underlying genetic and epigenetic mechanisms by which the quasi-UFP (Q-UFP, i.

Does the amount of formal care affect informal care? Evidence among over-60s in France.

This paper investigates the causal effect of the amount of formal care used on the informal care received by formal care users. We use an original instrument for formal care volume based on local disparities (NUTS 3 level, 96 units) in the price of formal care. Using the French CARE survey, we use a two-part model to assess the effect of formal care on the extensive and the intensive margin of informal care.