Publications by authors named "E P Ritter"

Here, we investigated the molecular genetic basis of mite domatia, structures on the underside of leaves that house mutualistic mites, and intraspecific variation in domatia size in Vitis riparia (riverbank grape). Domatia and leaf traits were measured, and the transcriptomes of mite domatia from two genotypes of V. riparia with distinct domatia sizes were sequenced to investigate the molecular genetic pathways that regulate domatia development and intraspecific variation in domatia traits.

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Across the tree of life, specialized structures that offer nesting sites to ants or mites - known as domatia - have evolved independently hundreds of times, facilitating ecologically important defence and/or nutritional mutualisms. Domatia show remarkable diversity in morphology and developmental origin. Here we review the morpho-anatomical diversity of domatia, aiming to unveil the primary mechanisms governing their development.

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Article Synopsis
  • A 39-year-old woman with a history of systemic lupus erythematosus (SLE) and antiphospholipid syndrome experienced ongoing symptoms including fever and joint pain.
  • She was admitted to the ER due to sudden difficulty breathing and discovered to have clubbed fingers, which started 5 months earlier; imaging showed severe mitral valve damage with a large mass.
  • Despite initial treatment for suspected infectious endocarditis and subsequent surgeries, the mass was ultimately diagnosed as high-grade sarcoma, underscoring the need for careful interdisciplinary management in unusual medical cases.
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Background: Intraductal papillary mucinous neoplasms (IPMNs) have the potential to evolve into pancreatic adenocarcinoma (PDAC). While main-duct IPMNs (MD-IPMNs), involving the main pancreatic duct (MPD), are less common than side-branch IPMNs (SB-IPMNs) or mixed-type IPMNs (mixed-IPMNs), their malignant transformation potential is far greater. Controversy exists between different guidelines in terms of recommended management strategies.

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  • * Research showed that smokers, both with and without CP, have higher sweat chloride levels and lower CFTR expression, indicating impaired CFTR activity due to smoking.
  • * Exposure to cigarette smoke and associated heavy metals like cadmium harms pancreatic function by reducing fluid and bicarbonate secretion and increasing intracellular calcium and ATP depletion.
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