Publications by authors named "E Ettema"
An acute rise of plasma Na concentration associates with syndecan-1 shedding during hemodialysis.
Am J Physiol Renal Physiol
August 2020
Endothelial dysfunction (ED) contributes to the high incidence of cardiovascular events in patients undergoing hemodialysis. Syndecan-1 in the endothelial glycocalyx can be shed into the circulation, serving as a biomarker for ED. As Na is a trigger for glycocalyx shedding, we now tested whether hemodialysis, with higher dialysate Na concentrations, is associated with more syndecan-1 shedding compared with standard hemodialysis (SHD).
View Article and Find Full Text PDFIn the Netherlands, increasing attention is being paid to psychiatric patients with a request for termination of life. One discussion related to this is the role of the family. However, the experiences and wishes of the family regarding their involvement in dealing with a psychiatric patient's request for termination of life, are not yet well documented.
View Article and Find Full Text PDFBackground: Intradialytic hypotension is a common complication of hemodialysis. The Hemocontrol biofeedback system, improving intradialytic hemodynamic stability, is associated with an initial transient increase in plasma sodium levels. Increases in sodium could affect blood pressure regulators.
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- Copeptin is being used more frequently as a substitute for vasopressin in epidemiological studies, but how renal function affects their levels and the copeptin/vasopressin ratio is not well understood.
- In a study involving 127 patients with varying stages of chronic kidney disease (CKD) and healthy participants, it was found that lower renal function resulted in higher levels of both copeptin and vasopressin, but the ratio between them behaved differently depending on the severity of kidney impairment.
- The findings suggest that copeptin can reliably substitute for vasopressin in individuals with better kidney function (eGFR >28 ml/min), but corrections are needed for those with severe CK
Hemodialysis is associated with a fall in myocardial perfusion and may induce regional left ventricular (LV) systolic dysfunction. The pathophysiology of this entity is incompletely understood, and the contribution of ultrafiltration and diffusive dialysis has not been studied. We investigated the effect of isolated ultrafiltration and isovolemic dialysis on myocardial perfusion and LV function.
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