Publications by authors named "Dusica Basaric"
Changes in the hemostatic system during COVID infection lead to hypercoagulability. Numerous studies have evaluated hemostatic abnormalities in COVID patients during acute infection, in the period of hospitalization. However, the hemostatic status following hospital discharge has not been sufficiently assessed.
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- Elevated factor VIII is an independent risk factor for deep venous thrombosis and pulmonary embolism, especially when combined with other risk factors.
- A study of 441 patients showed that factor VIII levels begin to rise in individuals over 40, reaching levels close to the thrombosis risk threshold of 1.5 IU/mL.
- Age significantly impacts factor VIII activity, while other comorbidities (except thyroid disease and malignancy) do not affect these levels in relation to thrombosis risk.
Background: Coagulation dysfunction represents a serious complication in patients during the COVID-19 infection, while fulminant thrombotic complications emerge as critical issues in individuals with severe COVID-19. In addition to a severe clinical presentation, comorbidities and age significantly contribute to the development of thrombotic complications in this disease. However, there is very little data on association of congenital thrombophilia and thrombotic events in the setting of COVID-19.
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