Study of a Narrow Fabric-Based E-Textile System-From Research to Field Tests.

Electronic textiles (e-textiles) are a branch of wearable technology based on integrating smart systems into textile materials creating different possibilities, transforming industries, and improving individuals' quality of life. E-textiles hold vast potential, particularly for use in personal protective equipment (PPE) by embedding sensors and smart technologies into garments, thus significantly enhancing safety and performance. Although this branch of research has been active for several decades now, only a few products have made it to the market.

OFSETH: smart medical textile for continuous monitoring of respiratory motions under magnetic resonance imaging.

The potential impact of optical fiber sensors embedded into medical textiles for the continuous monitoring of the patient during Magnetic Resonance Imaging is presented. We report on two pure optical sensing technologies for respiratory movements monitoring - a macro bending sensor and a Bragg grating sensor, designed to measure the elongation due to abdominal and thoracic motions during breathing. We demonstrate that the two sensors can successfully sense textile elongation between, 0% and 3%, while maintaining the stretching properties of the textile substrates for a good comfort of the patient.

Optical fibre sensors embedded into technical textile for a continuous monitoring of patients under Magnetic Resonance Imaging.

The potential impact of optical fiber sensors embedded into medical textiles for the continuous monitoring of the patient during Magnetic Resonance Imaging (MRI) is presented. In that way, we report on several pure optical sensing technologies for pulse oximetry and respiratory movements monitoring. The technique for pulse oximetry measurement is known as NIRS (Near Infra-Red Spectroscopy) in a reflectance mode.

Moyamoya disease and Down syndrome: case report and review of the literature.

We present the case of a 29-year-old woman with Down syndrome who developed bilateral frontal ischemic stroke. Cerebral angiography demonstrated an occlusion of the both supraclinoid internal carotid arteries associated with dilated collateral vessels, consistent with moyamoya disease. We review the clinical and radiological features of moyamoya disease associated with Down syndrome and discuss a few major physiopathologic hypotheses to explain this association.

[Motor neuropathy, putamen necrosis and optic nerve atrophy after acute methanol poisoning].

Optic neuropathy and putaminal necrosis are the most common sequellae of methanol poisoning. We report a case in a patient with a chronic motor neuro(no)pathy in addition to these neurological complications. Peripheral nerve and spinal cord disorders, related to methanol poisoning, are uncommon and probably underestimated.

Neurological complications of intravenous immunoglobulin (IVIg) therapy: an illustrative case of acute encephalopathy following IVIg therapy and a review of the literature.

We report the case of a 73-year-old man who developed an acute encephalopathy during IVIg therapy for AIDP. The signs and symptoms of the encephalopathy completely resolved after discontinuation of the treatment. We also reviewed the literature over the major neurological complications of IVIg therapy, including aseptic meningitis, cerebral infarction, and acute encephalopathy.

[Folic acid deficiency with leukoencephalopathy and chronic axonal neuropathy of sensory predominance].

A 44-year-old, alcoholic, woman developed a prolonged folate deficiency with a chronic predominantly axonal sensory polyneuropathy. The cerebral CT and MRI revealed a diffuse, clinically asymptomatic, leukoencephalopathy. A partial improvement of the neurological symptoms was observed after two months of oral administration of folate.

[Acute encephalomyopathy and persistent cerebellar syndrome after lithium salt and haloperidol poisoning].

A 44-year-old maniacodepressive woman developed acute encephalopathy due to the association of lithium carbonate and haloperidol. She was treated with lithium salts for many years and the serum level of lithium was within the therapeutic range. The encephalopathy was worsened by hyperthermia, dehydration, and reintroduction of haloperidol, 5 days after the first discontinuation of the neuroleptic.

Delayed-onset hemidystonia and chorea following contralateral infarction of the posterolateral thalamus. A case report.

A 68 year-old man developed progressive hemidystonia and chorea 8 months after a contralateral thalamic stroke. The neurological examination also showed a right pyramidal syndrome without hemiparesis, a right horizontal sectoranopia, and a right hemihypesthesia for all sensory modalities. The MRI revealed infarctions in the left medial temporo-occipital lobes and left posterolateral thalamus, corresponding to the vascular territories of both the thalamo-geniculate and posterolateral choroidal arterial pedicles.

[Cerebral cysticercosis treated by albendazole: development of cerebral magnetic resonance imaging].

A 36-year-old Indian man presented with a generalized tonic-clonic seizure. The diagnosis of neurocysticercosis was suspected by the presence of multiple cystic and nodular cerebral lesions on MRI, and was confirmed by serological studies (ELISA). He was treated with albendazole during one week at the daily dose of 15 mg/kg.

[Agenesis of the corpus callosum, heterotopia of the gray cortex and interhemispheric cyst. Late radiologic diagnosis in an asymptomatic adult].

Agenesis of the corpus callosum may occur as an isolated finding; most often, it is present in association with other cerebral malformations such as cortical heterotopias. Computerized tomography and magnetic resonance imaging are methods of choice to detect these cerebral prenatal abnormalities, especially asymptomatic forms. We report the case of an asymptomatic young adult with complete callosal agenesis, cortical heterotopia and interhemispheric cyst, on CT and MR imaging.

Neurotrophic ulcer following Wallenberg's syndrome.

A 61-year-old man consulted for two long-standing ulcerations on the scalp and one on the nose, following an acute vascular disorder of the brain stem (Wallenberg's syndrome). The aetiology and pathogenesis of neurotrophic ulcers are briefly discussed.

[Central pontine myelinolysis with cerebellar ataxia and dystonia].

A 46-year-old alcoholic woman presented with dementia, chronic cerebellar ataxia and diffuse dystonia. Neurological examination also demonstrated emotional lability and a pyramidal signs on the four limbs. CT showed mild cerebellar and frontal atrophy.

[Borrelia burgdorferi encephalomyelitis].

We report two patients with chronic encephalomyelitis due to Borrelia burgdorferi in whom the definite diagnosis was delayed because of atypical clinical features. The first patient presented with chronic spastic paraparesis, slight ataxia and nystagmus of several years' duration. A tentative diagnosis of multiple sclerosis was made in spite of important abnormalities of the CSF biological characteristics.

Lymphocytic meningoradiculitis and encephalomyelitis due to Borrelia burgdorferi: a clinical and serological study of 18 cases.

Clinical features of 18 patients with either lymphocytic meningoradiculitis (n = 17) or chronic encephalomyelitis (n = 1) due to Borrelia burgdorferi are reported. Arthropod bites were recorded in only seven patients. High titres of anti B burgdorferi antibodies were detected in sera from all patients and in CSF from 12 out of 17 patients tested.

C-reactive protein in serum and cerebrospinal fluid in various neurological disorders. Apparent local consumption during bacterial meningitis.

The level of C-reactive protein (CRP) was determined in the cerebrospinal fluid (CSF) by particle counting immunoassay. In non-neurological patients (N = 24), CRP was detectable only in 10 samples at concentrations ranging from 1.5 to 37 micrograms/l.

Immune complexes in cerebrospinal fluid and serum of neurological patients. Possible intrathecal formation in bacterial meningitis and herpetic encephalitis.

We assayed immune complexes (IC) by Particle Counting ImmunoAssay in the serum and cerebrospinal fluid (CSF) of patients with various neurological disorders. In pyogenic meningitis, the levels of IC sharply increased 4-8 days after onset with a fall before the 10th day of the disease. In herpetic encephalitis the IC and antibody levels started to increase about 12 days after onset.

The concentration of IgM in the cerebrospinal fluid of neurological patients.

The level of IgM was determined by Particle Counting Immunoassay in the cerebrospinal fluid. In non-neurological patients (N = 20) the mean was 97.5 micrograms/l with the upper reference limit at 380 micrograms/l.