Objective: To explore the mechanism of the p38MAPK signaling pathway in repairing articular cartilage defects with biological collagen membranes.
Methods: Thirty-two healthy adult male rabbits were randomly divided into a control group (n = 8), model group (n = 8), treatment group (n = 8) and positive drug group (n = 8). The control group was fed normally, and the models of bilateral knee joint femoral cartilage defects were established in the other three groups.
A novel ZSM-5 membrane with a low Si/Al ratio and homogeneous aluminum spatial distribution was achieved from an organic template-free inorganic gel in the presence of both OH(-) and F(-) ions and the obtained ZSM-5 membrane exhibited excellent selectivity and high flux and stability for dehydration of acetic acid in a wide AcOH content range.
View Article and Find Full Text PDFA thin tubular CAU-1 membrane of 2-3 μm exhibiting a high CO2 permeance of up to 1.34 × 10(-6) mol m(-2) s(-1) Pa(-1) and a CO2-N2 selectivity of 17.4-22.
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