The study focused on how tissue elements in the stomach's gastric mucosa adapt when influenced by both the bacteria's genetic traits and the host's genes.
Researchers collected venous blood and biopsy samples from young patients to analyze the health of their gastric mucosa and the genetic polymorphisms of certain interleukin genes.
Key findings indicated that specific bacterial strains led to increased cell turnover and changes in mucosal protein production, with certain genetic haplotypes being more common in patients a year after treatment.