Publications by authors named "D J J Halley"

Article Synopsis
  • * Post-mortem examinations of WTEs showed no major visible issues, but microscopic analyses revealed organ damage and virus presence, indicating a widespread infection.
  • * Phylogenetic studies indicate the HPAIV H5N1 genotype was likely introduced to Norway by migratory birds and was linked to outbreaks in seabirds, suggesting WTEs may act as indicators for these viruses, though not all strains seem to affect them similarly.
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Firefly flashes are well-known visual signals used by these insects to find, identify, and choose mates. However, many firefly species have lost the ability to produce light as adults. These "unlighted" species generally lack developed adult light organs, are diurnal rather than nocturnal, and are believed to use volatile pheromones acting over a distance to locate mates.

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Ants and other eusocial insects emit and receive chemical signals to communicate important information within the colony. In ants, nestmate recognition, task allocation, and reproductive distribution of labor are largely mediated through the detection of cuticular hydrocarbons (CHCs) that cover the exoskeleton. With their large size and limited volatility, these CHCs are believed to be primarily detected through direct contact with the antennae during behavioral interactions.

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Article Synopsis
  • - The study investigates the magnetic properties of epitaxial clusters made of chromium and chromium-vanadium oxides through various techniques, revealing that these clusters exhibit a small magnetic moment with super-paramagnetic behavior.
  • - The magnetization arises primarily from a unique magnetic phase of chromium oxide, contrasting with the bulk form of CrO, which is usually anti-ferromagnetic.
  • - Analysis of the magnetic properties at different temperatures suggests that the magnetic dipoles observed may be associated with short-range coupling between chromium and vanadium ionic spins, resembling the behavior in one-dimensional spin chains.
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Goldberg-Shprintzen syndrome (GOSHS) is caused by loss of function variants in the kinesin binding protein gene (KIFBP). However, the phenotypic range of this syndrome is wide, indicating that other factors may play a role. To date, 37 patients with GOSHS have been reported.

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