Publications by authors named "D Feldmann"

Article Synopsis
  • The study investigates the link between elevated C-reactive protein (CRP) levels and cardiovascular risk in systemic sclerosis (SSc) patients, focusing on how CRP positivity may indicate chronic inflammation and increased atherosclerosis risk compared to those without CRP elevation.
  • Researchers categorized 65 SSc patients into CRP+ (with prolonged elevated CRP levels) and CRP- (without elevated levels) to study associations with cardiovascular health, using methods like blood tests and carotid ultrasound to assess arteriosclerotic burden.
  • The goal is to better understand which cardiovascular and SSc-specific factors can differentiate patients with high CRP levels from those without, employing advanced classification techniques like random forest modeling
View Article and Find Full Text PDF

Hypothesis: Biphilic surfaces, namely surfaces comprising hydrophilic areas with a (super)hydrophobic background, are used in nature and engineering for controlled dropwise condensation and liquid transport. These, however, are highly dependent on the surface temperature and subcooling.

Experiments: Here, biphilic surfaces were cooled inside a rotatable environmental chamber under controlled humidity.

View Article and Find Full Text PDF

Fluid flows between rotating concentric cylinders exhibit two distinct routes to turbulence. In flows dominated by inner-cylinder rotation, a sequence of linear instabilities leads to temporally chaotic dynamics as the rotation speed is increased. The resulting flow patterns occupy the whole system and sequentially lose spatial symmetry and coherence in the transition process.

View Article and Find Full Text PDF

Objectives: About 25% of patients with systemic sclerosis (SSc) have elevated C-reactive protein (CRP) levels. Specific causes of CRP elevation are unknown so far. We aimed to investigate whether inflammatory arthritis is associated with CRP elevation.

View Article and Find Full Text PDF

Predisposition to anterior cruciate ligament (ACL) rupture is multi-factorial, with variation in the genome considered a key intrinsic risk factor. Most implicated loci have been identified from candidate gene-based approach using case-control association settings. Here, we leverage a hypothesis-free whole genome sequencing in two two unrelated families (Family A and B) each with twins with a history of recurrent ACL ruptures acquired playing rugby as their primary sport, aimed to elucidate biologically relevant function-altering variants and genetic modifiers in ACL rupture.

View Article and Find Full Text PDF