Publications by authors named "D E Popov"
Article Synopsis
- The study investigates CP symmetry violation in the decay of D^{+} particles into K^{-}K^{+}π^{+} using data from proton-proton collisions at a high energy of 13 TeV.
- A unique model-independent method was employed to analyze the phase-space distributions of D^{+} and D^{-} particles, correcting for any instrumental biases using D_{s}^{+} decays.
- The findings indicate no significant evidence of CP violation, with a p value of 8.1%, and measure specific CP asymmetry observables, marking this study as the most sensitive search of its kind in multibody decays.
Introduction: Previous studies have suggested an association between beta-cell and autonomic function and metabolic-associated fatty liver disease (MAFLD). We explored the association between controlled attenuated parameter (CAP) and insulin secretion and action, as well as sympathetic and parasympathetic activity in normal (NGT) and impaired (IGT) glucose tolerance.
Research Design And Methods: Twenty-five NGT (age 44.
to develop and validate two clinical simulation guides to support and disseminate a culture of safety in the surgical environment. methodological study supported by the International Nursing Association for Clinical Simulation and Learning theoretical framework. The snowball technique was used to validate the clinical guidelines with experts on the subject.
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- * A case study describes a 41-year-old man with malignant melanoma who developed acute paraplegia due to a fracture in his spine caused by a metastatic lesion.
- * Emergency surgery successfully relieved his spinal compression, leading to a quick recovery in motor and sensory functions, emphasizing the importance of early diagnosis and treatment in such cases.
Article Synopsis
- * The study used a modified cell line containing the m.15059G>A mutation to assess the impacts of eliminating this mutation on mitochondrial function.
- * Results showed that removing the m.15059G>A mutation enhanced mitochondrial membrane potential and efficiency, reduced harmful byproducts, and did not alter the antioxidant system, indicating the mutation's negative role in mitochondrial health.