Clinical simulation on intimate partner violence in adolescent girls: contributions to health teaching.

Objective: to learn about the contributions of using clinical simulation with undergraduate health students to care for adolescent girls in situations of intimate partner violence in the context of Primary Health Care.

Method: a qualitative study with 30 medical and 28 nursing undergraduates from a public university in Brazil. Data collection was mediated by holistic debriefing, with subsequent reflective and inductive thematic analysis.

Drinking pattern and sex modulate the impact of ethanol consumption on the mouse gut microbiome.

Gut microbiota impacts host homeostasis and diseases. Chronic plus binge ethanol consumption has been linked to increased injuries than chronic or binge ethanol intake alone. We hypothesized that distinct shapes in gut microbiota composition are induced by chronic, binge, and the association of these treatments, thereby affecting host functions and contributing to sex-based differences in alcohol use disorders.

Melatonin Prevents Thymic Atrophy but Does Not Protect Against Disruption of T Cell Maturation Related to Cyclophosphamide Exposure.

Increased oxidative stress and apoptosis are key mechanisms of thymic atrophy induced by cyclophosphamide (CYP). Atrophy leads to changes in the thymic microenvironment and disrupts T cell maturation. The hormone melatonin displays antioxidant and antiapoptotic effects.

Brazilian nurses in air transport of wounded during World War II.

Objective: to analyze the preparation of nurses from the Brazilian Expeditionary Force to work in the air transport of wounded people during World War II.

Method: historical-social study, of a qualitative nature, based on the work entitled "Nurses with the FAB on the Italian front: 1944-1945", written by nurse Izaura Barbosa Lima. The data results from a bibliographic survey, the use of documentary sources and database consultations, with the material being treated using Thematic Content Analysis.

IL-1 Receptor Contributes to the Maintenance of the Intestinal Barrier via IL-22 during Obesity and Metabolic Syndrome in Experimental Model.

Intestinal permeability and bacterial translocation are increased in obesity and metabolic syndrome (MS). ILC3 cells contribute to the integrity of intestinal epithelium by producing IL-22 via IL-1β and IL-23. This study investigates the role of IL-1R1 in inducing ILC3 cells and conferring protection during obesity and MS.