[The role of OCT in diagnosis and follow-up of diabetic macular ederma].

Purpose: To present the major importance of optical coherence tomography in early diagnosis and follow-up of diabetic macular edema.

Method: The patients diagnosed with diabetic retinopathy in our clinic between april 2008 - march 2011 were evaluated by OCT and other complementary exams.

Results: OCT in diabetic maculopathy is useful in the evaluation of the following: presence and area of retinal thickening; quantification of retinal thickening; proximity of retinal thickening to the centre of the macula; presence or absence of cyst formation (cystoid macular edema).

[Usher syndrome].

We present the case report of two brothers, PF-21 years old and PN-19 years old, to whom the fundus examination, perimetry and dark adaptation established the diagnosis of Retinitis Pigmentosa. The otorhinolaryngology exam and the audiogram revealed, in both cases, bilateral sensorineural deafness. The simultaneous presence of these two conditions completes the clinical findings of Usher syndrome.

[Sturge Weber syndrome in association with oculo-dermal melanosis].

We present the case report of a 10 years old patient to whom, based on the clinical examination, lab and imaging studies we established the diagnosis. Sturge- Weber syndrome, oculo-dermal melanocytosis-right side. This case is interesting because of the unusual and rare association of these two diseases.

[Sympathetic ophthalmia--clinical case].

This paper presents a case of Sympathetic ophthalmia following a penetrating ocular injury produced 30 days ago, of a patient who postponed the presentation to the ophthalmologist.

[Doyne retinal dystrophy--case report].

We present the case of a female patient, 16-years old, whose the fundus examination revealed the presence, in the macular region, of drusen type lesions; the clinical aspect completed with AFG, perimetry, dark adaptation, ERG resulted in the diagnosis of Doyne's honeycomb retinal dystrophy. The disorder, included in the Familial drusen, is due to an inborn error of metabolism localized the RPE. It seems that the defect is in an intercellular matrix protein which leads to the development of abnormal basement membranes.