Publications by authors named "Claudia Rezende Vieira de Mendonca Souza"
KPC-2 producing Pseudomonas putida as an unexpected pathogen of catheter-associated bloodstream infection.
J Infect Dev Ctries
April 2020
Article Synopsis
- Multidrug resistant Gram-negative bacteria, like Pseudomonas putida, are major global health threats, often leading to high mortality in bloodstream infections (BSIs).
- A case report from Brazil details a non-fatal peripheral line associated BSI in a 72-year-old man caused by P. putida with the blaKPC-2 carbapenemase gene.
- The study reveals that P. putida can transmit resistance genes, raising concerns about the spread of these resistant pathogens in healthcare and community settings.
High rate of detection of OXA-23-producing Acinetobacter from two general hospitals in Brazil.
Rev Soc Bras Med Trop
September 2019
Introduction: In recent decades, the prevalence of carbapenem-resistant Acinetobacter isolates has increased, and the production of oxacillinase (OXA)-type carbapenemases is the main mechanism underlying resistance. We evaluated OXA production from 114 Acinetobacter isolates collected between March and December 2013 from different clinical specimens of patients in two hospitals (Hospital 1 [n = 61] and Hospital 2 [n = 53]) located in Niterói, Rio de Janeiro, Brazil. We also evaluated the genetic diversity of OXA-producing isolates.
View Article and Find Full Text PDFIntroduction: Acinetobacter spp. have emerged as notorious pathogens involved in healthcare-associated infections. Carbapenems are important antimicrobial agents for treating infections due to multidrug resistant Acinetobacter spp.
View Article and Find Full Text PDFCarbapenemase production is an important mechanism of carbapenem resistance among nonfermentative Gram-negative isolates. This study aimed to report the detection of bla(OXA-58) gene in multiresistant clinical isolates of Acinetobacter baumannii recovered from inpatients in a public hospital. Polymerase chain reaction tests were performed to detect the bla(OXA-23-like), bla(OXA-24-like), bla(OXA-58-like) and bla(OXA-51-like) genes.
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