Publications by authors named "Christiane Teixeira Cartelle"
Spontaneous T. gondii neuronal encystment induces structural neuritic network impairment associated with changes of tyrosine hydroxilase expression.
Neurosci Lett
January 2020
Two billion people are chronically infected with Toxoplasma gondii worldwide with unknown consequences. Important neurological diseases have been associated to the brain infection, making essential to understand the neurophysiological changes associated with the neuronal encystment. T.
View Article and Find Full Text PDFBackground: In addition to the risk of developing ventilator-induced lung injury, patients with ARDS are at risk of developing hyperoxic injury due the supra-physiological oxygen supplementation clinically required to reverse hypoxemia. Alterations of endogenous surfactant system participate in the pulmonary dysfunction observed in ARDS. Administration of exogenous surfactant could have protective effects during hyperoxia.
View Article and Find Full Text PDFArticle Synopsis
- - This study investigates how a high-fat diet and mechanical ventilation impact inflammation in C57BL/6 mice by dividing them into four different groups based on diet and ventilation treatment over 10 weeks.
- - The results showed that mice on a high-fat diet had increased body weight and fat tissue, with specific inflammatory markers in the lungs elevated in groups that experienced either mechanical ventilation or both diet and ventilation.
- - Overall, the findings suggest that both obesity and mechanical ventilation contribute to increased lung inflammation and oxidative stress in these mice, highlighting the interplay between diet and respiratory treatment effects.
Correction: Enteric Neuronal Damage, Intramuscular Denervation and Smooth Muscle Phenotype Changes as Mechanisms of Chagasic Megacolon: Evidence from a Long-Term Murine Model of Tripanosoma cruzi Infection.
PLoS One
April 2017
Article Synopsis
- This text refers to a correction made to an article published under DOI: 10.1371/journal.pone.0153038.
- Corrections are typically needed to address errors or omissions found after the initial publication.
- The specific details of the correction are not provided in this excerpt.
Enteric Neuronal Damage, Intramuscular Denervation and Smooth Muscle Phenotype Changes as Mechanisms of Chagasic Megacolon: Evidence from a Long-Term Murine Model of Trypanosoma cruzi Infection.
PLoS One
August 2016
We developed a novel murine model of long-term infection with Trypanosoma cruzi with the aim to elucidate the pathogenesis of megacolon and the associated adaptive and neuromuscular intestinal disorders. Our intent was to produce a chronic stage of the disease since the early treatment should avoid 100% mortality of untreated animals at acute phase. Treatment allowed animals to be kept infected and alive in order to develop the chronic phase of infection with low parasitism as in human disease.
View Article and Find Full Text PDFObjectives: Different methods for the classification of leprosy have been proposed since the 1930s. The aim of this study was to compare the current methods at a referral center in Brazil.
Methods: The World Health Organization (WHO) operational classification was compared to the Ridley and Jopling classification, the Madrid classification, and a classification based on the number of body areas affected by skin and/or neural lesions (NBAA).
Defense against HSV-1 in a murine model is mediated by iNOS and orchestrated by the activation of TLR2 and TLR9 in trigeminal ganglia.
J Neuroinflammation
January 2014
Background: Herpes simplex 1 (HSV-1) causes various human clinical manifestations, ranging from simple cold sores to encephalitis. Innate immune cells recognize pathogens through Toll-like receptors (TLRs), thus initiating the immune response. Previously, we demonstrated that the immune response against HSV-1 is dependent on TLR2 and TLR9 expression and on IFN gamma production in the trigeminal ganglia (TG) of infected mice.
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