MRI evaluation of not complicated Tailgut cyst: Case report.

Introduction: The Tailgut cyst (cystic hamartoma) is an uncommon congenital disease of presacral retrorectal space and is embryologically part of some forms of enteric cysts. It is a benign malformation, although cases have been described in neoplastic degeneration.

Presentation Of Case: A caucasian 24 year old female presented shortly after childbirth with hypogastric abdominal discomfort associated with rectal tenderness, bleeding and moderate urinary symptoms for about three weeks.

Occult large epiphyseal solitary plasmacytoma at multidetector row computer tomography detected by magnetic resonance imaging.

Myeloma or Kahler-Bozzolo disease represents around 1% of all malignant tumors and 10% of the haematological variety; it is a B-lymphocellular malignant neoplasia which originates from plasma cells that produce monoclonal immunoglobulin, infiltrating in and destroying the adjacent bone tissue. Myeloma may be distinguished at radiological imaging in four distinct types: single osseous lesions (solitary plasmacytoma), diffused skeletal effects (myelomatosis), diffused osteopenia and sclerosing myeloma. It is known that initial osteolysis may not be shown through radiographic examination or CT; the lysis only becomes evident when there is a bone loss of over 50%, usually in the presence of a > or = 0.

Mangafodipir-DPDP enhanced MRI visualization of a pancreatic adenocarcinoma previously undetected by extracellular contrast enhanced CT and MRI.

We report a case of adenocarcinoma of the head of the pancreas, occult at extracellular contrast enhanced MDCT and magnetic resonance imaging (MRI), which was detected by MRI only with the use of a tissue-specific contrast agent (Mangafodipir trisodium Mn- DPDP). The histological examination after duodenopancreatectomy confirmed the diagnosis. Contrast-enhanced multi-detector computed tomography (MDCT) is currently considered to be the reference method for diagnosing and staging of pancreatic adenocarcinoma.

Pathogenesis and evolution of carpal instability: imaging and topography.

Carpal instability is a biomechanical alteration with a multiple pathogenesis which, if not identified and treated in time, leads to gradual articular collapse. Traumatism is known to be one of the main causes of carpal instability, while deposits of microcrystals caused by metabolic (chondrocalcinosis and gout) and congenital (ulna minus variance) diseases are less frequently involved in the pathogenesis. In forms secondary to traumatism, the trauma causes ligamentous injuries that lead to misalignments of the joint surfaces, or badly healed fractures with consequent articular incongruency.