The Contribution of Beverages to Intakes of Energy and MyPlate Components by Current, Former, and Never Smokers in the United States.

Background: Although beverage intake patterns have been shown to differ by smoking status, it is unknown whether the contributions of beverages to intakes of energy and MyPlate components also differ.

Objective: The purpose of this study was to compare beverage intakes and contributions of energy and MyPlate components by source (food alone, beverages alone, and food and beverages together) in diets of adult current, former, and never smokers.

Design And Participants: Dietary data from 4,823 men and 4,672 women aged ≥20 years who participated in What We Eat in America, National Health and Nutrition Examination Survey 2005-2008, were analyzed.

Impulsivity and Fast-Food Consumption: A Cross-Sectional Study among Working Adults.

Background: Little is known about the decision-making process of adults who choose to eat at fast-food restaurants. Impulsivity is the concept that individuals value immediate rewards and disregard future costs.

Objective: To determine the association between impulsivity and consumption of fast food among employed adults and to explore their reasons for eating fast food.

Food insecurity is associated with poor sleep outcomes among US adults.

Background: Although food insecure (FI) adults are at risk of chronic conditions, little research attention is given to their health behaviors, such as sleep.

Objective: We examined the associations between adult food security status and sleep duration, sleep latency, and sleep complaints reported to a health care professional.

Methods: Our population-based sample included 5637 men and 5264 women (≥22 y) who participated in the NHANES 2005-2010.

The pancreas-brain axis: insight into disrupted mechanisms associating type 2 diabetes and Alzheimer's disease.

Epidemiological and observational studies indicate a positive correlation between type 2 diabetes (T2DM) and dementia, with an increased risk of dementia and Alzheimer's disease (AD) associated with insulin-treated diabetes patients. The purpose of this review is to reveal the molecular mechanisms that connect physiological and pathological processes commonly observed in T2DM and AD. Conformational modifications in peptide residues, such as amyloid-β peptide in AD and amylin in T2DM have been shown to instigate formation of insoluble protein aggregates that get deposited in extracellular spaces of brain and pancreatic tissue thus disrupting their normal function.