Publications by authors named "Birtic K"

Aim: To prospectively evaluate long-term clinical outcome in patients who underwent radiofrequency catheter ablation for the treatment of idiopathic ventricular tachycardia (VT).

Methods: Twenty consecutive patients with idiopathic VT resistant to drugs were treated by temperature-controlled radiofrequency ablation. The site of VT origin was localized by pace mapping and endocardial activation mapping during VT.

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Unlabelled: The value of nonfunctional infrahisal second-degree atrioventricular (AV) block induced by incremental atrial pacing was prospectively examined in 192 patients with chronic bundle branch block (BBB) and syncope. We compared 174 (91%) patients with normal response to atrial pacing (Group I) to 18 (9%) patients with atrial pacing induced nonfunctional infrashisal second-degree AV block (Group II). Patients in group I had higher incidence of organic heart disease, ventricular tachycardia induction, and retrograde ventriculoatrial conduction (P < 0.

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In 71 patients with a myocardial infarction (MI) (anterior in 27, inferior in 44 patients) global (GEF) and regional (REF) left ventricular ejection fractions were determined by radionuclide ventriculography and estimated from a 12 lead electrocardiogram (ECG), using Selvester's QRS score, during the early phase of a MI (15 to 21 days following MI). Global ejection fractions determined by radionuclide ventriculography and from ECG using Palmeri's method were: for all MI 40.8 +/- 12.

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Quantitative and qualitative analyses of Q waves and QRS scores were performed on 69 patients during the early phase of first myocardial infarction (MI) and 6 months subsequently. The regression of ECG signs of MI were compared with the enzymatically estimated size of MI, the location of MI, and with the changes of global ejection fraction (GEF) assessed by radionuclide ventriculography. Among 57 patients with Q wave MI a complete disappearance of ECG signs of MI was found in 9 (15.

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In 60 patients myocardial infarction size was determined by electrocardiogram (ECG) using Selvester's QRS scoring system. These values were compared with the size of infarction as determined enzymatically using gram equivalent isoenzyme MB creatine kinase (gEq) and using maximum values of isoenzyme MB-CK. The results showed no statistically significant difference between the size of anterior and inferior infarction determined by gEq (25.

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