Publications by authors named "Bingting Zhuo"
Article Synopsis
- The study investigates how air pollution impacts chronic respiratory disease (CRD) risk by analyzing metabolomic data from over 171,000 participants without COPD or asthma.
- Researchers found that higher exposure to particulate matter and nitrogen oxides correlated with increased CRD incidence, identifying specific metabolites linked to these pollutants.
- The results suggest that certain metabolic processes, specifically inflammatory and erythrocyte-related pathways, mediate the effects of air pollution on CRD risk.
Background: Studies have reported the associations between inflammation, brain volume, and cognition separately. It is reasonable to assume peripheral inflammation may contribute to cognitive decline through brain volume atrophy.
Objective: To examine the associations between peripheral inflammation, brain volume, and cognition among adults, and to investigate whether brain volume atrophy mediates the inflammation-cognition relationshipMethods:We retrieved 20,381 participants with available data on peripheral inflammation, brain volume, and cognition from the UK Biobank cohort.
We aimed to estimate the spatiotemporal trends in the global burden of chronic obstructive pulmonary disease (COPD) attributable to both household air pollution from solid fuels (HAP) and ambient particulate matter (APM) from 1990 to 2019 and compared the possible differences between the burdens attributable to APM and HAP. The number of deaths, disability-adjusted life-years (DALYs), and years of life lost (YLLs) of COPD attributable to HAP from solid fuels and APM during 1990-2019 were extracted from the Global Burden of Diseases Study 2019. The proportion of YLLs in DALYs and average YLLs per COPD death were also calculated.
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