The () gene encodes the plasma membrane Na/H antiporter, SOS1, that is mainly responsible for extruding Na from the cytoplasm and reducing the Na content in plants under salt stress and is considered a vital determinant in conferring salt tolerance to the plant. However, studies on the salt tolerance function of the gene of recretohalophytes, such as , are limited. In this work, the effects of salt stress on cotton seedlings transformed with tobacco-rattle-virus-based virus-induced gene silencing (VIGS) of the endogenous gene, or strain K599-mediated -transgenic hairy root composite cotton plants exhibiting VIGS of were first investigated.
View Article and Find Full Text PDFBackground: The anionic toxicity of plants under salt stress is mainly caused by chloride (Cl). Thus Cl influx, transport and their regulatory mechanisms should be one of the most important aspects of plant salt tolerance studies, but are often sidelined by the focus on sodium (Na) toxicity and its associated adaptations. Plant chloride channels (CLCs) are transport proteins for anions including Cl and nitrate (NO), and are critical for nutrition uptake and transport, adjustment of cellular turgor, stomatal movement, signal transduction, and Cl and NO homeostasis under salt stress.
View Article and Find Full Text PDFThe gene was previously demonstrated to have high water channel activity by its heterologous expression in oocytes and in yeast; it also plays a significant role in growth of transgenic plants under favorable conditions and has enhanced tolerance toward salt and drought treatment. In this work, we first investigated the physiological effects of heterologous expression in soybean cotyledon hairy roots or composite plants mediated by toward enhanced salt tolerance. The transgenic soybean plants mediated by the pollen tube pathway, represented by the lines N and J11, were analyzed at the physiological and molecular levels for enhanced salt tolerance.
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