From boundaries to bumps: When closed (extremal) contours are critical.

Invariants underlying shape inference are elusive: A variety of shapes can give rise to the same image, and a variety of images can be rendered from the same shape. The occluding contour is a rare exception: It has both image salience, in terms of isophotes, and surface meaning, in terms of surface normal. We relax the notion of occluding contour and, more accurately, the rim on the object that projects to it, to define closed extremal curves.

Colour, contours, shading and shape: flow interactions reveal anchor neighbourhoods.

Two dilemmas arise in inferring shape information from shading. First, depending on the rendering physics, images can change significantly with (even) small changes in lighting or viewpoint, while the percept frequently does not. Second, brightness variations can be induced by material effects-such as pigmentation-as well as by shading effects.

Model cortical association fields account for the time course and dependence on target complexity of human contour perception.

Can lateral connectivity in the primary visual cortex account for the time dependence and intrinsic task difficulty of human contour detection? To answer this question, we created a synthetic image set that prevents sole reliance on either low-level visual features or high-level context for the detection of target objects. Rendered images consist of smoothly varying, globally aligned contour fragments (amoebas) distributed among groups of randomly rotated fragments (clutter). The time course and accuracy of amoeba detection by humans was measured using a two-alternative forced choice protocol with self-reported confidence and variable image presentation time (20-200 ms), followed by an image mask optimized so as to interrupt visual processing.

Human-mediated foot-and-mouth disease epidemic dispersal: disease and vector clusters.

Disease clusters were retrospectively explored at national level using a geo-referenced dataset from the 2001 Uruguayan Foot-and-Mouth Disease (FMD) epidemic. Disease location and time (first 11 epidemic weeks) were analysed across 250 counties (of which 160 were infected), without and with control for human mobility related factors (human population and road densities). The null hypothesis of random disease distribution over space and/or time was assessed with: (i) purely temporal; (ii) purely spatial; and (iii) space/time tests.