[Bacteremia caused by Lactobacillus plantarum in endocarditis lenta].

A report is presented on a 43-year-old patient with a subacute endocarditis due to an immunovasculitis and a bloodstream infection caused by Lactobacillus plantarum. The causative agent was isolated six times from blood cultures. L.

Contributions of C1q, bacterial lipopolysaccharide, and porins during attachment and ingestion phases of phagocytosis by murine macrophages.

In contrast to the S-form of Salmonella minnesota, its Re mutant binds to mouse peritoneal macrophages. The binding reaction triggers an oxidative burst, measured by a chemiluminescent reaction. The oxidative burst was abolished in the presence of either purified lipopolysaccharide or porins (outer membrane proteins) extracted from the Re mutant, suggesting that both components are involved in binding of the Re mutant to macrophages.

Comparison of the exoproducts of gram-negative bacteria by SDS-Page.

The protein exoproducts released during exponential growth of Gram-negative bacteria were analysed and compared by sodium dodecyl sulfate-polyacrylamide gel electrophoresis (SDS-Page). The following bacterial strains were tested: Escherichia coli, Klebsiella pneumoniae, Citrobacter freundii, Enterobacter cloacae, Serratia liquefaciens, Serratia rubidaea, Proteus mirabilis, Proteus vulgaris, Salmonella minnesota, Pseudomonas aeruginosa and Pseudomonas fluorescens. It is demonstrated by SDS-Page that members of one species show identical protein pattern, whereas different species show besides comparable protein bands a species characteristic pattern.