Publications by authors named "Andrea Gallorini"

Methylmercury (MeHg), a neurotoxic pollutant, is formed mainly under anaerobiosis. The "Modulo Sperimentale Elettromeccanico" (MOSE), built to temporarily close the Venice Lagoon and protect the city from flooding, induces changes in the hydrological regime, reducing water circulation and decreasing in the dissolved oxygen concentrations of the lagoon. Our study shows the potential changes in sediment and overlying water physico-chemistry in a simulated MOSE closing-event by incubating sediment cores for 48 h in the laboratory and deploying benthic chambers.

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Article Synopsis
  • Dreissena polymorpha, a bivalve commonly found in freshwater ecosystems, shows promise as a biomonitor due to its efficiency in filtering and absorbing toxins, but its molecular stress responses to contaminants, particularly when exposed to multiple pollutants, are not well understood.
  • Research found that co-exposure to carbamazepine (CBZ) and methylmercury (MeHg) led to significant changes in protein and metabolite levels after 24 and 72 hours, highlighting specific alterations in neurotransmission pathways.
  • The study demonstrated that co-exposure caused more molecular changes than single exposures, emphasizing the combined toxic effects of CBZ and MeHg, and stressing the need for further investigation into molecular toxicity pathways in D. polymorpha
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Hg methylation in the oxic water column of marine environments has been linked to the presence of suspended and settling particles known as marine snow, which acts as a micro-niche for MeHg production. While marine snow has been thoroughly studied, its freshwater counterpart, lake snow, received less attention, even though few works have highlighted its ability to be a micro environment for Hg methylation in freshwater systems. Here we present new data of MeHg and THg concentrations in the lake snow of a deep peri-alpine lake (Lake Geneva, Switzerland-France).

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Carbamazepine (CBZ) and Hg are widespread and persistent micropollutants in aquatic environments. Both pollutants are known to trigger similar toxicity mechanisms, e.g.

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