Differential Phenolic Compounds and Hormone Accumulation Patterns between Early- and Mid-Maturing Sweet Cherry ( L.) Cultivars during Fruit Development and Ripening.

Color acquisition is one of the most distinctive features of fruit development and ripening processes. The color red is closely related to the accumulation of polyphenolic compounds, mainly anthocyanins, during sweet cherry fruit maturity. In non-climacteric fruit species like sweet cherry, the maturity process is mainly controlled by the phytohormone abscisic acid (ABA), though other hormones may also play a role.

RNAseq reveals different transcriptomic responses to GA in early and midseason varieties before ripening initiation in sweet cherry fruits.

Gibberellin (GA) negatively affects color evolution and other ripening-related processes in non-climacteric fruits. The bioactive GA, gibberellic acid (GA), is commonly applied at the light green-to-straw yellow transition to increase firmness and delay ripening in sweet cherry (Prunus avium L.), though causing different effects depending on the variety.

Effects of Water Deficits on Growth, Water Status and Stomata Functioning.

The effect of water deficit on growth, water status and stomatal functioning of was investigated under controlled water conditions. The study was done at the Antumapu Experiment Station of the University of Chile. Three levels of water stress were tested: (i) well-watered (WW), (ii) medium stress intensity (low-watered (LW)) and (iii) intense stress (non-watered (NW)), with 10 replicates each level.

Gibberellic Acid Modifies the Transcript Abundance of ABA Pathway Orthologs and Modulates Sweet Cherry () Fruit Ripening in Early- and Mid-Season Varieties.

Several phytohormones modulate ripening in non-climacteric fruits, which is triggered by abscisic acid (ABA). Gibberellins (GAs) are present during the onset of ripening in sweet cherry fruits, and exogenous gibberellic acid (GA) application delays ripening, though this effect is variety-dependent. Although an ABA accumulation delay has been reported following GA treatment, the mechanism by which GA modulates this process has not been investigated at the molecular level in sweet cherry.