[Ocular involvement in patient with Wegener's granulomatosis--a case report].

Wegener's granulomatosis is a systemic vasculitis. Most often granulomatous inflammation is situated in upper and lower respiratory tracts as well as kidneys. Ocular involvement affects 50-60% of patients with Wegener's granulomatosis.

[The new serological markers of rheumatoid arthritis].

Rheumatoid arthritis (RA) is the most frequent human chronic inflammatory autoimmune disease. Its etiology is up to date not known. RA has been proven to be an antigen-specific lymphocyte T driven process.

[Graft-versus-host disease as the cause of symptoms mimicking Sjögren's syndrome].

A case of chronic graft-versus-host disease (chronic GvHD) mimicking symptoms associated with idiopathic Sjögren's syndrome is presented. Hypotheses on the pathophysiological origin of clinical syndromes associated with graft-versus-host disease are discussed.

[Diagnostic difficulties in polymyositis].

Polymyositis is a connective tissue disease. Although myositis is the dominant clinical manifestation, internal organs may also be affected. Arthritis occurs in 30% of patients, especially in the course of the anti-synthetase syndrome.

[Coincidence of juvenile idiopathic arthritis and multiple sclerosis: case report].

Introduction: Juvenile idiopathic arthritis is a systemic pathology of connective tissue characterized by a chronic inflammatory process with an autoimmune background whereas multiple sclerosis is a demyelination disease with an important role of immune disorders in its pathogenesis. The etiology in both cases remains unknown. The coincidence of juvenile idiopathic arthritis and multiple sclerosis was described a just a few patients.

[Polymyositis and systemic sclerosis symptoms as a clinical manifestation of a chronic graft versus host disease: a case report and literature review].

A case of a patient presenting with polymyositis and systemic sclerosis symptoms due to chronic graft versus host disease is reported. The literature review about similar disease manifestations and hypotheses regarding the pathophysiological origin of the clinical syndromes combined with graft versus host reaction is presented.