Sphenocentrum jollyanum (Pierre) aqueous leaf extract demonstrates anti-inflammatory and mitochondrial-restorative influences while modulating apoptosis in streptozotocin-induced diabetic rats.

Ethnopharmacological Relevance: Sphenocentrum jollyanum (Pierre) is a medicinal plant native to West African countries, especially Nigeria and Ghana. The leaf of S. jollyanum is a traditional therapy for diabetes, erectile dysfunction, gastrointestinal disorders, and malaria.

Influences of (turmeric) supplemented diet on the expression of defensive and destructive factors in indomethacin-induced ulcerated Wistar rats.

Background: Turmeric ( has been recognized as a plant with high medicinal value and it has been used in the prevention and treatment of many diseases. Different studies have shown that turmeric has both therapeutic and preventive effects on peptic ulcer. However, there are controversial reports about the anti-ulcerogenic potential of turmeric.

Defensive potential and deleterious impact of turmeric (Curcuma longa) L rhizome powder supplemented diet on antioxidant status of indomethacin-induced ulcerated wistar rats.

Effects of various concentration of turmeric (Curcuma longa [C longa]) rhizome powder (TRP) supplemented diet on the antioxidant status of selected organs (stomach, liver, kidney, and heart) of indomethacin (Indo)-induced ulcerated wistar rats were investigated in this study. The rats were fed with different percentages of TRP supplemented diet (1% [A], 2% [B], 5% [C], and 10% [D]) for 28 days. It was observed from the results that animals fed on 10% turmeric rhizome supplemented and those in the ulcerogenic group had significant reduction in kidney, cardiac gluthione (GSH), superoxide dismutase (SOD), catalase (CAT), and gastric GSH when compared with normal control animals and other pre-treated groups.

Effect of low-zinc status and essential fatty acids deficiency on the activities of aspartate aminotransferase and alanine aminotransferase in liver and serum of albino rats.

The effects of dietary deficiencies of zinc and essential fatty acids (EFAs) or both on aspartate aminotransferase (AST) and alanine aminotransferase (ALT) were investigated in young growing rats. Four groups of albino rats were fed diets deficient in either EFA (4% hydrogenated coconut oil) or zinc (6 ppm) or both. The control diet was adequate in EFA (4% soybean oil) and zinc (100 ppm).