Publications by authors named "A Koutsoukis"

Anomalous aortic origin of a coronary artery (AAOCA) is a rare congenital anomaly with a large spectrum of anatomical variations. Selective engagement of an AAOCA can present challenges during cardiac catheterization. A comprehensive understanding of the characteristics of major AAOCA can effectively assist operators for selecting and maneuvering catheters.

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Article Synopsis
  • The study examines the occurrence and location of coronary artery disease (CAD) in adults with anomalous aortic origin of a coronary artery (AAOCA), which is not well documented.
  • Researchers analyzed data from 390 patients, focusing on the presence of CAD through imaging techniques like coronary angiography.
  • The findings revealed that CAD is more prevalent in the proximal segment of retroaortic AAOCA compared to other types of AAOCA, with no CAD found in the proximal segment of interarterial AAOCA, and the reasons for these differences remain unclear.
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Anomalous aortic origin of the coronary arteries are congenital anomalies with many anatomical forms. Due to the varying risk of sudden death, these abnormalities must be classified accurately. There are still questions about the mechanism and individual risk of sudden death, the natural history of these abnormalities and the benefits of a surgical correction.

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A patient with severe, symptomatic functional mitral regurgitation was initially considered not suitable for MitraClip (Abbott Vascular, Abbott Park, Illinois) implantation because of non-coapting mitral leaflets. Repeated levosimendan infusions in combination with intensive diuresis induced sufficient valve coaptation, thus allowing MitraClip implantation to be performed. ().

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Background: Patients with high-gradient (HG) severe aortic stenosis (AS) and left ventricular (LV) dysfunction are at high risk of death. The optimal timing for aortic valve replacement (AVR) is not defined by guidelines. The objective was to define the optimal timing to perform isolated AVR in patients with HG-AS and severe LV dysfunction.

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