Effective Hamiltonians for Rapidly Driven Many-Body Lattice Systems: Induced Exchange Interactions and Density-Dependent Hoppings.

We consider 1D lattices described by Hubbard or Bose-Hubbard models, in the presence of periodic high-frequency perturbations, such as uniform ac force or modulation of hopping coefficients. Effective Hamiltonians for interacting particles are derived using an averaging method resembling classical canonical perturbation theory. As is known, a high-frequency force may renormalize hopping coefficients, causing interesting phenomena such as coherent destruction of tunneling and creation of artificial gauge fields.

Intrinsic photoconductivity of ultracold fermions in optical lattices.

We report on the experimental observation of an analog to a persistent alternating photocurrent in an ultracold gas of fermionic atoms in an optical lattice. The dynamics is induced and sustained by an external harmonic confinement. While particles in the excited band exhibit long-lived oscillations with a momentum-dependent frequency, a strikingly different behavior is observed for holes in the lowest band.

Directed transport in a classical lattice with a high-frequency driving.

We analyze the dynamics of a classical particle in a spatially periodic potential under the influence of a periodic in time uniform force. It was shown by S. Flach and coworkers [Phys.

Fermi acceleration in time-dependent rectangular billiards due to multiple passages through resonances.

We consider a slowly rotating rectangular billiard with moving boundaries and use canonical perturbation theory to describe the dynamics of a billiard particle. In the process of slow evolution, certain resonance conditions can be satisfied. Correspondingly, phenomena of scattering on a resonance and capture into a resonance happen in the system.

A novel human-specific soluble vascular endothelial growth factor receptor 1: cell-type-specific splicing and implications to vascular endothelial growth factor homeostasis and preeclampsia.

A human-specific splicing variant of vascular endothelial growth factor (VEGF) receptor 1 (Flt1) was discovered, producing a soluble receptor (designated sFlt1-14) that is qualitatively different from the previously described soluble receptor (sFlt1) and functioning as a potent VEGF inhibitor. sFlt1-14 is generated in a cell type-specific fashion, primarily in nonendothelial cells. Notably, in vascular smooth muscle cells, all Flt1 messenger RNA is converted to sFlt1-14, whereas endothelial cells of the same human vessel express sFlt1.