CD69 Plays a Beneficial Role in Ischemic Stroke by Dampening Endothelial Activation.

Rationale: CD69 is an immunomodulatory molecule induced during lymphocyte activation. Following stroke, T-lymphocytes upregulate CD69 but its function is unknown.

Objective: We investigated whether CD69 was involved in brain damage following an ischemic stroke.

Long-term fluctuation of standard automatic perimetry, pulsar perimetry and frequency-doubling technology in early glaucoma diagnosis.

Purpose: Analyze the stability and accuracy of 3 perimetric techniques.

Methods: A total of 104 stable eyes (65 subjects) with ocular hypertension and early glaucoma [group G, mean defect = 1.08 dB, SD = 2.

[Focal neuropsychological deterioration by posterior lobar atrophy (posterior cortical dementia)].

A right-handed 59 year old man who presented difficulties in the performance of complex acts (praxic) and spatial disorientation is described. Neurophysical exploration showed deterioration of the capacities linked to perceptive and spatial organization preserving the verbal capacities including the memory. After two years and a half of follow up practically no variation has been observed in his clinical manifestations with no implication of other superior mental function.

A prospective study of atrial fibrillation and stroke.

In a prospective study of 72 patients with stroke and atrial fibrillation, we classified strokes as cardioembolic or noncardioembolic based on arterial assessment using Doppler sonography and angiography. We analyzed and cross-tabulated 18 clinical characteristics and found four to be significantly associated with a cardioembolic mechanism: stroke with onset during activity and peak deficit at onset (p less than 0.008), previous infarct in a different vascular territory (p less than 0.