Publications by authors named "A F Abdel-Fattah"
Background: Carbapenem-resistant (CRE) infections pose a significant global public health threat. We aimed to assess the risk variables, clinical characteristics, and outcomes of CRE-caused infections in criticalcare patients.
Patients And Methods: This prospective study enrolled 181 adult patients infected with in the intensive care unit (ICU).
: Patients with cirrhosis who seem normal during physical examinations may still have abnormalities in their electroencephalogram (EEG) or show pathological results in neuropsychological tests. This study aimed to investigate the progression of minimal hepatic encephalopathy, its effects on quality of life, its prognostic value, and its significance for daily functioning. : This study involved 50 patients with confirmed cirrhosis (28 Child A, 12 Child B, 10 Child C) who were assessed for psychological symptoms and underwent several tests: the Minimal Mental State Examination (MMSE), the Letter Cancellation Test, the Digit Symbol Coding Test, and EEG.
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- A 71-year-old man with symptoms like headaches and vision problems was diagnosed with a pituitary fossa lesion and thyroid cancer that had metastasized to his brain.
- Imaging tests revealed a significant tumor affecting multiple areas, and a biopsy confirmed the presence of metastatic papillary adenocarcinoma.
- The study includes a literature review of similar cases to understand the clinical features, treatment, and outcomes related to thyroid cancer spreading to the pituitary gland.
Introduction Ankle fractures are common yet debilitating injuries. Surgery is the mainstay treatment for ankle fractures displaying a high suspicion of instability. During the coronavirus disease 2019 (COVID-19) pandemic, conservative management became prominent in patients with ankle fractures.
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- - The study investigates how cellular interactions, driven by force, contribute to cancer cell invasion, particularly in the context of cerebral cavernous malformations (CCM), which are characterized by abnormal blood vessels.
- - Researchers used an in-vitro model to demonstrate that endothelial cells lacking the CCM2 protein help recruit normal (wild-type) endothelial cells through mechanical forces and changes in the surrounding extracellular matrix, facilitating lesion growth.
- - The findings reveal that CCM2 mutant cells manipulate neighboring wild-type cells into proliferating and altering their functions, providing new insights into the mechanisms behind vascular abnormalities and tools for studying cell behavior in disease contexts.