"A place to call home": A process of liberation for Rohingya Refugees in the United States.

This study explores the experiences of Rohingya refugees in the United States following their forced migration and years as stateless refugees. Qualitative strengths-based phenomenological analysis was applied to individual interviews with eight key informants, all identified as Rohingya refugees. Interviews focused on experience and meaning-making concerning the complexities of Rohingya identity and belonging, rights and liberation in the United States, and the impact of generational, gender, and migration patterns.

Gain-of-Function Chromatin Remodeling Activity of Oncogenic FOXL2-C134W Reprograms Glucocorticoid Receptor Occupancy to Drive Granulosa Cell Tumors.

Adult type ovarian granulosa cell tumors (AGCTs) are rare malignancies with the near universal c.C402G (p.Cys134Trp) somatic mutation in FOXL2, a Forkhead box-family transcription factor important for ovarian function.

Aberrant expression of collagen type X in solid tumor stroma is associated with EMT, immunosuppressive and pro-metastatic pathways, bone marrow stromal cell signatures, and poor survival prognosis.

Background: Collagen type X (ColXα1, encoded by ) is expressed specifically in the cartilage-to-bone transition, in bone marrow cells, and in osteoarthritic (OA) cartilage. We have previously shown that ColXα1 is expressed in breast tumor stroma, correlates with tumor-infiltrating lymphocytes, and predicts poor adjuvant therapy outcomes in ER/HER2 breast cancer. However, the underlying molecular mechanisms for these effects are unknown.

promoter mutations and survival outcomes in adult-type granulosa cell tumors.

Objectives: To evaluate survival outcomes among patients with adult-type granulosa cell tumors who have telomerase reverse transcriptase () promoter mutations.

Methods: This is a retrospective cohort study using the MD Anderson Rare Gynecologic Malignancy Registry. Patients with adult granulosa cell tumors who underwent molecular testing for promoter and c.