[Survival in primary dilated cardiomyopathy as a function of tobacco smoking. A retrospective study].

In order to determine the influence of tobacco smoking on the course and long-term prognosis of idiopathic dilated cardiomyopathy, we conducted a retrospective study of 111 patients (95 men and 16 women, mean age 45.5 +/- 8.1 years) who had undergone cardiac catheterization between January 1970 and December 1979 and had been followed up for 6 to 16 years.

Dilated cardiomyopathy: long-term follow-up and predictors of survival.

To determine long-term survival and the prognostic factors of dilated cardiomyopathy, we retrospectively studied a consecutive series of 111 patients (95 men, 16 women, mean age: 45.5 +/- 8.1 years) undergoing cardiac catheterization and diagnostic coronary angiography from January 1970 to December 1979.

[Correlation between late surface potentials and inducible ventricular arrhythmias. Apropos of 115 cases].

Programmed ventricular stimulation and a search for late surface potentials (LP) by means of signal-averaging and quantification by Simson's method were performed in 115 patients divided into five groups: group I (n = 50) with no myocardial infarction (MI) and no clinical or induced ventricular tachycardia (VT); group II (n = 28) with MI and no clinical or induced VT; group III (n = 8) with MI, no clinical VT but induced non sustained VT (NSVT); group IV (n = 19) with MI, no clinical VT but induced sustained VT (SVT); group V (n = 15) with clinical and induced VT. The terminal portion of the averaged signal (AS) was analyzed by three parameters: duration of AS terminal portion less than 40 microvolts (Dur TP), AS amplitude 50 and 40 ms before its end (Amp 50, Amp 40). The presence of TP was defined in two ways: criterion A = Amp 40 less than 25 mcv, criterion B = at least two sub-criteria: Dur TP greater than 40 ms, Amp 50 less than 25 mcv, Amp 40 less than 15 mcv.